Diabetes: Natural Treatments That Really Work!

By George John Georgiou

Diabetes is a serious disease that normally requires taking medications for life. This book will allow you to use a powerful and scientifically proven natural treatment protocol to completely cure your diabetes. Learn what you need to do to be diabetes free forever. Also learn why you have diabetes and what you need to do to reverse it now. Ther

• Language: English
• Publisher: Da Vinci Health Publishing
• Release date: Feb 5, 2019
• ISBN: 9789925569076

George John Georgiou

Dr. Georgiou, Ph.D.,D.Sc (AM).,N.D., is a chartered biologist, iridologist, naturopath, herbalist, homeopath, nutritionist, bioresonance specialist, acupuncturist, clinical psychologist and clinical sexologist. He has been a clinician most of his life and is the Director Founder of the Da Vinci Holistic Health Center in Larnaca, Cyprus (www.naturaltherapycenter.com) which specializes in the natural treatment of chronic diseases, heavy metal toxicity and Candidiasis, along with many other health problems. He is also the Founder Director of the Da Vinci Institute of Holistic Medicine (www.collegenaturalmedicine.com) as well as the Da Vinci BioSciences Research Center. He is the author of 23 books, a clinician and researcher.

Book preview

Diabetes:

Natural Treatments

that Really Work!

Dr George J Georgiou, Ph.D.,N.D.,D.Sc (AM)

Dedication

First, I would like to bow deeply to the thousands of patients who have helped me understand the complexities of this disease process and who need to take credit for the time spent with them in refining this protocol over many years. 

All these patients over the years have been my laboratory for developing many treatment protocols through trial and error, backed by research.

I would also like to thank all the researcher scientists, lecturers and teachers who dedicate their life to helping others, and all the courageous health professionals who go against the grain of the establishment, while thinking outside the box.

A loving hug of gratitude to my wife and 4 children for their support and understanding during my professional endeavours throughout these years – they are all blessed.

Finally, I deeply embrace the Divine faith that I have been blessed with, that has helped me believe in the innate healing abilities of the body, through the power of Natural healing, without chemical intervention.

A profound blessing to you all and may your healing journey be fruitful and fulfilling!

Copyright © 2018 Dr. George J. Georgiou. All rights reserved. No portion of this book, except for brief review, may be reproduced, stored in a retrieval system, or transmitted in any form or by any means—electronic, mechanical, photocopying, recording, or otherwise—without the written permission of the publisher.

For information contact Da Vinci Health Publishing – [e-mail].

Published by:

Da Vinci Health Publishing 

Panayia Aimatousa 300

Aradippou 7101

Larnaca

Cyprus

MEDICAL DISCLAIMER: The following information is intended for general information purposes only. Individuals should always see their health care provider before administering any suggestions made in this book. Any application of the material set forth in the following pages is at the reader’s discretion and is his or her sole responsibility.

ISBN - 978-9925-569-07-6

Contents

Chapter 1: Diabetes: Natural Approaches to Healing

Chapter 2: The Holistic Model of Health

Chapter 3: Toxicity: Underlying Cause of All Diseases

Chapter 4: Detoxification: The Health Secret of all Time

Chapter 5: Food Intolerances, Inflammation and Disease

Chapter 6: Candida: A Universal Cause of Many Diseases

Chapter 7: Curing with Energetic Medicine and Bioresonance

Chapter 8: Emotional, Psychological & Spiritual Roots of Disease

Disclaimer

Summary and Concluding Remarks

ABOUT THE AUTHOR

More Books written by Dr Georgiou:

Chapter 1: Diabetes: Natural Approaches to Healing

Introduction

Diabetes is a serious, chronic disease that occurs either when the pancreas does not produce enough insulin (a hormone that regulates blood sugar, or glucose), or when the body cannot effectively use the insulin it produces (WHO 2017). Diabetes is an important public health problem, one of four priority noncommunicable diseases (NCDs) targeted for action by world leaders. Both the number of cases and the prevalence of diabetes have been steadily increasing over the past few decades.

Diabetes of all types can lead to complications in many parts of the body and can increase the overall risk of dying prematurely. Possible complications include heart attack, stroke, kidney failure, leg amputation, vision loss and nerve damage. In pregnancy, poorly controlled diabetes increases the risk of foetal death and other complications.

From my clinical experience dealing with Type 2 diabetes, it is possible to greatly improve and even heal this disease with the correct natural approach and diet. This cannot be achieved without some serious lifestyle changes – mainly dietary - that need to be adhered to for life.

Remember, 2,500 years ago, Hippocrates, the Father of Medicine, said to his students, Let thy food be thy medicine and thy medicine be thy food.

Moses Maimonides, the great 12th century physician, repeated the Hippocratic statement when he said, No illness which can be treated by diet should be treated by any other means.

In essence, Hippocrates and Maimonides were insisting that their students practice nutrition therapy; healing the body not by pills, drugs, or surgery, but by changing what you eat. This type of medical therapy is being used by doctors today, but only by a very small minority.

Prevalence and Key Facts

Here are some key facts about diabetes given by the WHO:

The number of people with diabetes has risen from 108 million in 1980 to 422 million in 2014.

The global prevalence of diabetes among adults over 18 years of age has risen from 4.7% (1980) to 8.5% (2014).

Diabetes prevalence has been rising more rapidly in middle- and low-income countries.

Diabetes is a major cause of blindness, kidney failure, heart attacks, stroke and lower limb amputation.

In 2012, an estimated 1.5 million deaths were directly caused by diabetes and another 2.2 million deaths were attributable to high blood glucose.

Almost half of all deaths attributable to high blood glucose occur before the age of 70 years. WHO projects that diabetes will be the 7th leading cause of death in 2030.

Healthy diet, regular physical activity, maintaining a normal body weight and avoiding tobacco use are ways to prevent or delay the onset of type 2 diabetes.

Diabetes can be treated and its consequences avoided or delayed with diet, physical activity, medication and regular screening and treatment for complications.

Fig. 1 looks at the prevalence of diabetes globally. An amazing 1 in 11 adults have diabetes (2015), which represents nearly 10% of the entire population. Moreover, one in two adults still remain undiagnosed. Western Pacific countries tend to have the highest prevalence (153.2 million) while Africa has the lowest rate (14.2 million).

Types of Diabetes

Type 1 diabetes (previously known as insulin-dependent, juvenile or childhood-onset diabetes) is characterized by deficient insulin production in the body. People with type 1 diabetes require daily administration of insulin to regulate the amount of glucose in their blood. If they do not have access to insulin, they cannot survive. This involves about 10% of all diabetics and a large number of these are children.

Symptoms include excessive urination and thirst, constant hunger, weight loss, vision changes and fatigue.

Fig 1. Prevalence of diabetes worldwide 2015 – 2040.¹

Type 2 diabetes (formerly called non-insulin-dependent or adult-onset diabetes) results from the body’s ineffective use of insulin. Type 2 diabetes accounts for the vast majority of people with diabetes around the world (WHO, 2017). This comprises the majority of diabetics – over 80%.

Symptoms may be like those of type 1 diabetes, but are often less marked or absent. Thus, the disease may go undiagnosed for several years, until complications have already arisen. For many years, type 2 diabetes was seen only in adults but it has begun to occur in children.

Impaired glucose tolerance (IGT) and impaired fasting glycaemia (IFG) are intermediate conditions in the transition between normal blood glucose levels and diabetes (especially type 2), though the transition is not inevitable. People with IGT or IFG are at increased risk of heart attacks and strokes.

Gestational diabetes (GDM) is a temporary condition that occurs in pregnancy and carries a long-term risk of type 2 diabetes. The condition is present when blood glucose values are above normal. Women with gestational diabetes are at increased risk of some complications during pregnancy and delivery, as are their infants. Gestational diabetes is diagnosed through prenatal screening, rather than reported symptoms. This comprises about 10% of all diabetic patients.

Symptoms of Diabetes

There are many symptoms of diabetes that are similar for both types (see Fig. 2 for a summary). The more common symptoms include:

increased urine output,

excessive thirst,

weight loss,

hunger,

fatigue,

skin problems

slow healing wounds,

yeast infections, and

tingling or numbness in the feet or toes.

How do I know if I have diabetes?

The early symptoms of untreated diabetes are related to elevated blood sugar levels and loss of glucose in the urine. High amounts of glucose in the urine can cause increased urine output (frequent urination) and lead to dehydration. This dehydration also causes increased thirst and water consumption.

Fig 2. Summary of Symptoms

Other symptoms include:

A relative or absolute insulin deficiency eventually leading to weight loss.

The weight loss of diabetes occurs despite an increase in appetite.

Some untreated diabetes patients also complain of fatigue.

Nausea and vomiting can also occur in patients with untreated diabetes.

Frequent infections (such as infections of the bladder, skin, and vaginal areas) are more likely to occur in people with untreated or poorly-controlled diabetes.

Fluctuations in blood glucose levels leading to blurred vision.

Extremely elevated glucose levels leading to lethargy and coma.

Risk Factors for Diabetes

There are many risk factors for type 2 diabetes and pre-diabetes. The following can increase your risk of developing type 2 diabetes. The Lancet has also published environmental risk factors for type 1 diabetes (Fig 3):

Family history of diabetes

Overweight

Elevated levels of triglycerides and low levels of good cholesterol (HDL)

Unhealthy diet

Physical inactivity

Increasing age

High blood pressure

Ethnicity

Polycystic Ovary Syndrome

Impaired glucose tolerance (IGT)*

Insulin resistance

History of gestational diabetes

Poor nutrition during pregnancy

Fig 3. Environmental risk factors for type 1 diabetes²

Diagnosing Diabetes

Normally, fasting glucose levels are 70-100mg/100ml (or mg%) of blood or 3.9-5.6mmol/l. One hour after a meal or a glucose load it should be less than 160mg% or 9.0mmol/l. If test results are considerably higher than normal, diabetes is diagnosed. Table 1 below shows the diagnostic criteria as laid out by the World Health Organization (WHO):

Table 1. Current WHO recommendations for the diagnostic criteria for diabetes and intermediate hyperglycaemia

Drugs Used for Treating Diabetes

I will not go into the detailed pharmacology of drugs as this will not really help – what we can look at briefly are some of the common groups of drugs that are commonly used to control diabetes, and how they work on the body.

Insulin is the most common type of medication used in type 1 diabetes treatment. It can also be used in type 2 diabetes treatment when blood sugar levels become incontrollable with oral medication. Insulin is given by injection into muscles and comes in different types. The type of insulin you need depends on how severe your insulin depletion is.

Let’s take a brief look at some of the groups of medications used by medical doctors and how they work in the body:

Alpha-glucosidase inhibitors

These medications help your body break down starchy foods and table sugar. This effectively lowers your blood sugar levels.

Biguanides

Biguanides decrease how much sugar your liver makes and also decrease how much sugar your intestines absorb, while making your body more sensitive to insulin, and helping your muscles absorb glucose. This is one of the most common forms of oral medication used.

DPP-4 inhibitors

DPP-4 inhibitors help the body continue to make insulin. They work by reducing blood sugar without causing hypoglycaemia (low blood sugar). These drugs can also help the pancreas make more insulin.

Glucagon-like peptides (incretin mimetics)

These drugs are similar to the natural hormone called incretin. They increase B-cell growth (the cells in the pancreas that actually produce insulin) and how much insulin your body uses. They decrease your appetite and how much glucagon your body uses. They also slow stomach emptying.

Meglitinides

These medications help your body release insulin. However, in some cases, they may lower your blood sugar too much.

Sulfonylureas

These are among the oldest diabetes drugs still used today. They work by stimulating the pancreas with the help of beta cells. This causes your body to make more insulin.

Thiazolidinediones

These medications work by decreasing glucose in your liver. They also help your fat cells use insulin better. These drugs come with an increased risk of heart disease. If your doctor gives you one of these drugs, they will watch your heart function during treatment.

All of these drugs have side-effects over time. The problem these days is that modern doctors are routinely giving insulin to people with type 2 diabetes simply because it reduces blood sugar levels.

The reality, though, is that type 2 diabetics who take insulin injections die at more than double the rate of those given non-insulin treatment

The study, Mortality and Other Important Diabetes-Related Outcomes With Insulin vs Other Antihyperglycemic Therapies in Type 2 Diabetes,³ investigated 84,622 primary care patients with type 2 diabetes from 2000 to 2010 and compared the results of these treatments. Basically, the main treatment using insulin had the most adverse effects. In fact, Insulin monotherapy resulted in:

2.0 times more myocardial infarctions

1.7 time more major adverse cardiac events

1.4 time more strokes

3.5 times more renal complications

2.1 times more neuropathy

1.2 times more eye complications

1.4 times more cancer

2.2 times more deaths

Modern medicine's hubris allows it to make claims that are simply not supported. Based on those unsupported claims, thousands - and in the case of diabetes, millions - of people are placed on drugs and regimens that have never been demonstrated to have any beneficial effect.

The use of insulin in type 2 diabetics is only one example, but it's been clearly demonstrated again and again with disasters like Vioxx.

Why Does Insulin Accelerate Morbidity and Mortality?

Now, a new study published in the Journal of Clinical Endocrinology & Metabolism titled, Insulin administration may trigger type 1 diabetes in Japanese type 2 diabetes patients with type 1 diabetes high-risk HLA class II and the insulin gene VNTR genotype, is shedding light on a possible explanation for why insulin treatment may accelerate morbidity and mortality in type 2 diabetics.

The study revealed that giving genetically susceptible type 2 diabetes patients recombinant insulin can trigger their bodies to target their own insulin producing cells for autoimmune destruction, effectively producing 'double diabetes': type 1 and type 2, as a result. Blood tests revealed that the patients had antibodies against their own pancreatic islet cells (the cells responsible for producing insulin), insulin allergy or increased levels of insulin antibody.

Complications of Diabetes

People with diabetes have an increased risk of developing a number of serious health problems. Consistently high blood glucose levels can lead to serious diseases affecting the heart and blood vessels, eyes, kidneys, nerves and teeth. In addition, people with diabetes also have a higher risk of developing infections. In almost all high-income countries, diabetes is a leading cause of cardiovascular disease, blindness, kidney failure, and lower limb amputation.

TYPE 1 DIABETES

Who Gets Type 1 Diabetes?

Only about 5% of people with diabetes have type 1. It’s more common in whites than in African-Americans. It affects men and women equally. Although the disease usually starts in people under 20, it can happen at any age.

Causes of Type 1 Diabetes

The main factor in Type 1 diabetes is clearly the destruction of the insulin-producing beta cells in the pancreas, due to an inflammatory autoimmune reaction. Diabetes may manifest only after 90% of the beta cells have been destroyed. Medical doctors do not really know the reasons – they call this idiopathic which literally means no known cause.

Researchers do know that genes play a role; there is an inherited susceptibility. However, something must set off the immune system, causing it to turn against itself and leading to the development of type 1 diabetes.

Genes Play a Role in Type 1 Diabetes

Some people cannot develop type 1 diabetes because they do not have the genetic coding that researchers have linked to type 1 diabetes. Scientists have figured out that type 1 diabetes can develop in people who have a particular HLA complex. HLA stands for human leukocyte antigen, and antigens function is to trigger an immune response in the body.

There are several HLA complexes that are associated with type 1 diabetes, and all of them are on chromosome 6.

Different HLA complexes can lead to the development of other autoimmune disorders, such as rheumatoid arthritis, ankylosing spondylitis, or juvenile rheumatoid arthritis. Like those conditions, type 1 diabetes has to be triggered by something - usually a viral infection.

What Can Trigger Type 1 Diabetes

When a virus invades the body, the immune system starts to produce antibodies that fight the infection. T cells are in charge of making the antibodies, and then they also help in fighting the virus.

However, if the virus has some of the same antigens as the insulin-producing cells in the pancreas - the beta cells - then the T-cells can actually turn against the beta cells. The T-cell products (antibodies) can destroy the beta cells, and once all the beta cells in your body have been destroyed, you cannot produce enough insulin.

It usually takes a number of years for the T cells to destroy the majority of the beta cells, but that original viral infection is what is thought to trigger the development of type 1 diabetes.

Not every virus can trigger the T-cells to turn against the beta cells. The virus must have antigens that are similar enough to the antigens in beta cells, and those viruses include:

B4 strain of the coxsackie-B virus (which can cause a range of illnesses from gastrointestinal problems to myocarditis - inflammation of the muscles of the heart)

German measles

Mumps

Rotavirus (which generally causes diarrhoea).

There have also been some controversial studies into the connection between drinking cow’s milk as an infant and the development of type 1 diabetes. Researchers do not all agree on this, but some believe that the proteins in cow’s milk are similar to a protein that controls T-cell production called glycodelin⁴. 

The baby’s body attacks the foreign protein - the cow’s milk protein - but then also attacks glycodelin, leading to an overproduction of T-cells. And too many T cells in the body can lead to those T cells destroying the pancreas’s insulin-producing beta cells.

Researchers have made significant progress in understanding the cause of type 1 diabetes, and they’re still hard at work to figure out why certain viruses trigger it and why T-cells turn against beta cells. The medical community wants to better understand the cases of diabetes in order to prevent it.

Other Causes of Type 1 Diabetes

When one thinks a little more holistically, there are several reasons that could explain this increasing epidemic, particularly amongst the younger generation. These factors would include:

food allergies – such as reactions to wheat and milk proteins

dysbiosis or overgrowth of pathogenic microbes in the intestines

vitamin B6 deficiency

toxic metals such as arsenic in food and water – arsenic is known to kill pancreas cells

The diabetic pancreas shows a greatly increased number of white blood cells of a type associated with allergies. If you cut out all food intake of a person diagnosed with Type 1 diabetes and put them on a fast of green apples or diluted lemon juice, their blood sugar levels often return to normal and may remain normal as long as only selected non-allergenic foods are used.

Food Triggers

It is well documented that in susceptible individuals, something in wheat known as gliadin stimulates diabetogenic class II HLA antigens on the surface of the pancreatic islet cells (cells that normally do no display these antigens), marking them for autoimmune destruction.

The triggers that can activate these genes are certain antigenic foods like wheat, cow's dairy and (unfermented, GMO and/or excessive) soy. The triggers that fire this loaded gun are varied, from prolonged exposure to these foods, to increased intestinal/gut permeability, vaccinations, viral infections, pesticide and chemical exposures and perhaps a multitude of unknown factors.

If indeed this is the case, then it would be wise to keep these two most commonly lauded health foods (cow's milk products and wheat) away from our children if they are indeed implicated in the development of type 1 diabetes.

If one can eradicate the causes of autoimmune self-destruction by treating infections, removing diabetogenic foods, correcting mineral and vitamin deficiencies and imbalances, changing the pH of the tissues back to their normal alkaline state and supplementing the diet with proven beta-cell regenerating foods, herbs, or nutrients, the pancreas (in some cases) can regenerate beta cell function.

Type 2 diabetes, and the pre-diabetic state of insulin resistance that precedes it, are caused by the following preventable factors:

Excessive consumption of empty calories.  

Inactivity; lack of exercise.

Consumption of hidden sweets, which are high-glycaemic foods that peak blood sugar levels such as pasta, cereals and crackers. 

Excess sugar, but even worse, high fructose corn syrup, and other sources of concentrated fructose such as agave.

Hydrogenated oils and trans fats.

Nutritional deficiencies of minerals, especially magnesium, chromium and zinc, and omega 3 fatty acids, as found in foods like flaxseed, walnuts and wild fish.

Chemical exposures, including environmental pollution, e.g. pesticides, and drugs, e.g. various prescribed and over the counter drugs.

There are hidden sweet foods that are added to many processed foods that will immediately spike blood sugar levels.

High Fructose Corn Syrup (HFCS) and purified fructose have both been shown to cause insulin resistance in rats and humans. There are, in fact, over 70 other adverse health effects linked to purified fructose consumption. Hydrogenated oils also dramatically reduce the responsiveness of our muscle and fat to insulin, whereas omega 3 fatty acids increase that responsiveness.  

There are also many other foods that contain preservatives that can all add to the body’s burden of toxicity, as well as increasing inflammation in the body.

Please note that many of these causative factors responsible for type 1 diabetes, can also be a cause of type 2 diabetes.

Sugars and refined carbohydrates – main culprits of diabetes!

In an excellent book, The Saccharine Disease, Dr. T. L. Cleave demonstrates that many of the diseases that presently afflict us are relatively modern plagues. His view, and what many other scientists around the globe believe, is that these diseases of civilization are due largely to the incredible increase in sugar consumption during the past century.

Cleave shows that many diseases that are common today were virtually unknown until the introduction of refined sugar. These conditions include constipation, diabetes, diverticular disease, varicose veins, thrombosis, haemorrhoids, dental caries (cavities), the twin plagues of obesity and diabetes, E. coli infections, and peptic ulcers. He also touches on the subject of colon cancer.

Cleave rules out the possibility that these diseases were caused by the refining of wheat, since the wide-scale use of white bread dated from 1800, while the onslaught of these common modem diseases only happened in the early 20th century.

It is hard to comprehend the massive increase in sugar use in our society. In 1815, the average resident of Great Britain consumed about 15 pounds of sugar per year. When Cleave's book was published in England in 1974, this had risen to about 120 pounds per year. In the United States in 1999, each person consumed about 158 pounds of sugar every year! That’s a pound of sugar every two to three days!

Related image

An influx of sugar into the bloodstream upsets the body's blood-sugar balance, triggering the release of insulin, which the body uses to keep blood-sugar at a constant and safe level. Insulin also promotes the storage of fat, so that when you eat sweets high in sugar, you're making way for rapid weight gain and elevated triglyceride levels, both of which have been linked to cardiovascular disease.

Complex carbohydrates tend to be absorbed more slowly, lessening the impact on blood-sugar levels. The reason free sugar has such an effect is because it is in its free form and not bound to anything, which explains the sudden rise in insulin after a sugary snack or drink. Fruits and vegetables do have sugar, but it is bound to a variety of vitamins, minerals, enzymes, and fibre which ensures a natural digestion and absorption process.

Our bodies were never meant to ingest sugar in its free form!

Understanding the link between sugar and diabetes

Although eating sugar is not directly linked to developing diabetes, some evidence suggests that increased overall availability of sugar makes diabetes more common. A 2013 study⁵ that looked at 175 different countries found that more sugar in the food supply increased diabetes rates.

Specifically, for every additional 150 calories of sugar available per day per person, diabetes levels rose 1 percent. This change continued even when researchers controlled other factors linked to diabetes, such as obesity, exercise, and overall calorie consumption.

This research suggests that sugar consumption does affect diabetes, at least on a population level.

The study did not look at individuals, so does not support the claim that individual sugar consumption causes diabetes. Despite this, it gives people who eat a lot of sugar something to consider, especially if they have other diabetes risk factors.

A 2012 review⁶ of previous research suggests that some forms of sugar consumption could increase the risk of diabetes. Drawing upon previous research, the study suggested that sugary drinks were likely to increase the risk of type 2 diabetes. Research on the link between other forms of sugar intake and diabetes, as well as sugar and other health risk factors, did not reach any firm findings

Let us know examine the importance of toxins and how these can play a role in the development of diabetes – both type 1 and type 2.

Environmental Toxins - Diabetogens

The incidence of diabetes has increased 7 to 10-fold in the past 50 y. Although increased sugar consumption, obesity, and lack of exercise certainly contribute, the effect of environmental toxins may be far greater. The data are so compelling that some researchers now label these toxins as diabetogens.

There certainly does appear to be a clear correlation between the levels of Persistent Organic Pollutants, abbreviated to POP’s in the environment and the incidence of diabetes.  POP’s would include PCB’s, polychlorinated biphenyls, DDE, dichlorodiphenyldichloroethylene, DDT, hexachlorocyclohexane, dichlorodiphenyltrichlorocyclohexane, and HCH.

The Table below⁷ clearly shows the literal parallel line between the increase in synthetic organic chemicals produced during the second world war and beyond, and the prevalence of diabetes.

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The research is telling us that phthalates, bisphenol A and PCB’s from plastics, and arsenic from water and food (seafood, rice, mushrooms, and poultry) seem to be the major diabetogens that are related to the increasing prevalence of diabetes.  The primary mechanism appears due to damaging pancreatic β cells with resultant decreased production of insulin.⁸

There are essentially 5 types of strategies for decreasing body load of toxins – we will talk about these in more detail in the treatments discussed later:

Avoidance (the best!).

Increasing glutathione production (facilitates phase 2 conjugation and helps protect from the oxidation and inflammation which facilitate the damage to blood sugar regulation).

Increasing dietary fiber (helps bind toxins in the gut to facilitate excretion from the body).

Toxin-specific interventions to increase detoxification/elimination.

Toxin-specific interventions to prevent the damage they cause.

Internal toxins can also target the pancreas

There are internal microbes that can produce toxins inside our own body that can cause a chronic inflammation in the pancreas. When the normal protective gut bacteria are under stress, pathogenic microbes will take over. Due to the inappropriate over-use of antibiotics, this is now very common.

Should these pathogens invade not only the lower part of the intestinal tract, but also the duodenum, (the upper part of the small intestine) then pancreatitis or inflammation of the pancreas may result. This has been demonstrated experimentally. It is not even necessary for microbes to invade the pancreas itself as their breakdown products or endotoxins do most of the damage.

Clearly, a low-grade chronic inflammation of the pancreas makes it a primary target organ for any allergic reaction.

Xanthurenic Acid and vitamin B6 deficiency

Another internal toxin produced by the body, can also damage the beta cells of the pancreas, leading to diabetes. This metabolite that our body produces is called xanthurenic acid and can accumulate to high levels if we are deficient in vitamin B6. When the levels of vitamin B6 are low in our body, the amino acid tryptophan cannot be properly metabolized, part of which is normally converted to niacin (vitamin B3) or nicotinamide.

Instead, an abnormal metabolite, xanthurenic acid, accumulates. High levels of this have been shown in animal experiments to damage the beta cells, and within days, such animals developed diabetes (Stone and Darlington, 2002).

The sooner the missing vitamin is supplied in high doses, the easier the blood sugar regulation could be normalized again. Individuals who are even mildly deficient in vitamin B6 excrete xanthurenic acid in the urine. This is used as a standard laboratory test for vitamin B6 deficiency. So it may be worth being tested for vitamin B6 deficiency, which is a simple urine test done in many labs these days.

If indeed your levels of xanthurenic acid are high, then taking magnesium, zinc and vitamin B6 supplements will reduce the formation of xanthurenic acid. These minerals are generally deficient in diabetics.

Another interesting fact is that high doses of nicotinamide (vitamin B3) can postpone the need for insulin injections in newly diagnosed Type I diabetics for months and even years. A high level of this B-vitamin inhibits the formation of xanthurenic acid from tryptophan, it protects the beta cells from an autoimmune attack, and makes the cellular energy production more efficient.

Now let us look at another connection between milk and diabetes that a lot of researchers have looked at and is therefore an important factor.

Connecting Milk and Gluten

Another line of investigation by various researchers has shown relationships between immune reactions and cow’s milk. A study by Laugeson et al (2003) looked at several hundred newly diagnosed diabetic children and showed that all of them had an immune response to a fragment of cows' milk protein. What’s more, this protein fragment had the same composition as one called P69 on the beta cells.

Juvenile diabetes is much higher in those who have been bottle-fed rather than breast-fed, and it is lower in communities that consume fewer cows' milk products. However, it appears that this protein fragment is only a problem with milk from Friesian cows (called A1 milk) but not with milk from other, lower-yielding breeds that produce A2 milk with a higher fat content.

Most cow’s milk consumed is A1 milk

The milk protein casein has different variations (A1 or A2) depending on the breed of cow. Laugesen and Elliott (2003) found that consumption of milk containing the A1 type of casein is associated with type 1 diabetes (breeds that produce A1 milk are more common in northern Europe, where type 1 incidence is higher). 

A2 milk usually comes from sheep, goats, donkey, camel, horse, buffalo, reindeer, yak and some breeds of cows such as Jersey, Guernsey and Asian.

Image result for type a1 cows

Because bottle-fed infants are very susceptible to colds, respiratory and gastrointestinal infections - six and more infections a year - they are more likely to receive antibiotics that then encourage overgrowth in the intestines of undesirable microbes, and a tendency to chronic pancreatitis.

One type of E. coli bacteria is harmless in the large intestine, but has the potential for causing severe damage in the small intestine. This is because it produces a molecule that is very similar to insulin. When the immune system becomes activated against this molecule, it may then also direct its attack against related features in the beta cells.

More recently it has also been shown that an autoimmune attack on the pancreas can be