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Medicine in Brief: Name the Disease in Haiku, Tanka and Art
Medicine in Brief: Name the Disease in Haiku, Tanka and Art
Medicine in Brief: Name the Disease in Haiku, Tanka and Art
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Medicine in Brief: Name the Disease in Haiku, Tanka and Art

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Medicine in Brief is a collection of poems in traditional Japanese haiku and tanka form. Each poem strives to capture the uniqueness of a particular disease, serving both as puzzle and springboard for education about the illness.
LanguageEnglish
PublisherXlibris US
Release dateFeb 6, 2022
ISBN9781669810001
Medicine in Brief: Name the Disease in Haiku, Tanka and Art
Author

Cynthia Cooper MD

Author profile: Dr. Cynthia Cooper is an award-winning medical educator at Harvard Medical School and a member of the Core Educator Faculty for the Department of Medicine at Massachusetts General Hospital. She is passionate about the use of creative approaches to medical education and making the complex world of Medicine accessible to all learners. She lives in Carlisle, MA with her husband and children. Illustrator profile: Dr. Pamela Chen is currently a Pediatrics resident at Boston Children’s Hospital and Boston Medical Center. Her medical interests include education, mentoring and diversity inclusion. Outside of the hospital, she spends her time painting and singing. She lives in Brookline, MA with her husband and mischievous cat.

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    Medicine in Brief - Cynthia Cooper MD

    Copyright © 2022 by Cynthia Cooper, MD.

    All rights reserved. No part of this book may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system, without permission in writing from the copyright owner.

    Any people depicted in stock imagery provided by Getty Images are models, and such images are being used for illustrative purposes only.

    Certain stock imagery © Getty Images.

    Rev. date: 02/02/2022

    Xlibris

    844-714-8691

    www.Xlibris.com

    837712

    These poems are stereotypes of diagnoses and cannot

    reflect the full spectrum of disease or patient experience.

    The explanations contain medical information but

    should not be a substitute for advice from a physician.

    They are here to serve as puzzles, mnemonics, and

    celebrations of the art of diagnosis in medicine.

    Dedicated to my husband who always knew I could.

    Tell me your story

    The exam augments the plot

    Together we learn

    CONTENTS

    Cardiopulmonary

    Infectious Disease

    Rheumatology

    Endocrinology

    Gastroenterology/Hepatology

    Nephrology

    Integumentary

    Gynecology/Genitourinary

    Hematology/Oncology

    Neurology/Psychiatry

    Acknowledgments

    Cardiopulmonary.jpg

    Cardiopulmonary

    Shinhai

    Transform blue to red

    Exchange, filter, rhythmic stead

    Ceaseless, not silent

    A dull ache, my jaw

    Winded with such small actions

    Clenched an iron fist

    Coronary Artery Disease

    A mismatch between the supply of oxygen and the demand of myocardial tissues leads to signs and symptoms of cardiac ischemia. The classic history is one of deep discomfort or pressure with exertion, often radiating to the jaw or shoulder. A fixed occlusion leads to a predictable pattern of exertion and pain. A dynamic occlusion presents as crescendo symptoms that may herald an impending myocardial infarction.

    Chest squeezed in a vise

    Pet a cat, now pay the price

    Must find a puffer

    Breath extended, whistling through

    Need quick act from beta-2

    Asthma

    This pulmonary disease is commonly diagnosed in childhood, but it can span age groups. Hallmarks are air flow restriction and reversible bronchoconstriction, often tied to an environmental or exertional trigger. Patients describe chest tightness, cough, and wheezing. Airways show inflammation. In mild cases, a short-acting inhaler with a beta-2 agonist is first line management.

    Drowning while in bed

    Must sit up, window, cool air

    Each breath a gurgle

    Congestive Heart Failure

    Acute congestive heart failure with an elevated left atrial pressure often presents with shortness of breath and pulmonary edema. Orthopnea, or upright breath, is increased dyspnea while lying down. Blood volume, held in the legs by gravity when the patient is standing, returns to the circulation and overwhelms a struggling, noncompliant heart. Paroxysmal nocturnal dyspnea is a similar symptom, waking the patient from sleep with acute shortness of breath and the desire to sit or stand up.

    One minute flies by

    With too little breath escaped

    Cachectic, pursed lips

    Diaphragms flatten, suck ribs

    Elbows dark from constant prop

    Emphysema

    Emphysema is a pulmonary disease characterized by flow limitation, notably decreased forced expiratory volume measured at one minute and end exhalation. Bleb formation, loss of surface area for gas exchange, and air trapping are notable features, leading to flattening of the diaphragm. The inward horizontal movement of the flattened diaphragm on the ribs is called the Hoover sign and highlights the loss of the diaphragm’s up and down movement for inhalation and exhalation. Patients will often position themselves in a tripod position, with elbows propped on a fixed surface, so that they can use their neck muscles to lessen the work of breathing. Most cases of emphysema are associated with a history of cigarette smoking, typically with apical lung involvement. Alpha-1 antitrypsin deficiency preferentially involves the base of the lungs.

    Stretched atria send

    Erratic syncopation

    AV node resists

    Atrial fibrillation

    Atrial fibrillation is the most common cardiac arrhythmia whose incidence increases with age. The sinoatrial node is overwhelmed by intra-atrial impulses, which are erratic, fast and fail to organize atrial contraction. The refractory period of the AV node serves as a point of resistance, preventing the atrial impulses from reaching the ventricle. Atrial fibrillation is a major risk factor for embolic stroke.

    Genes, stress, drugs, or salt

    Vessel tense, near to bursting

    Dull, constant assault

    Ceaseless, silent erosion

    Slowly wearing out the parts

    Essential Hypertension

    Nearly half the world’s adults have hypertension, and it is the most common reason for both office visits and use of chronic prescription meds. Hypertension is a multifaceted disease with the sympathetic nervous system, blood volume, and the renin-angiotensin-aldosterone system all playing a role. Age, obesity, family history, sodium and alcohol intake, and reduced nephron number all may influence blood pressure. Many drugs including oral contraceptives, NSAIDs, antidepressants, and corticosteroids can exacerbate existing hypertension. Heart failure, ischemic stroke, hemorrhagic stroke, and chronic kidney disease all are tied to poorly controlled blood pressure.

    Every breath sharp pained

    Pounding, full jugular veins

    Right heart panic strain

    Lungs on x-ray clear, air-filled

    POCUS D sign, tension build

    Acute Pulmonary Embolism

    Acute pulmonary embolism is the sudden, potentially life-threatening lodging of a blood clot into the pulmonary arterial vasculature. Clots typically form in the lower extremity in the setting of stasis, endothelial damage, or hypercoagulability. The clinical picture is one of acute dyspnea, pleuritic chest pain, tachycardia, and right heart pressure overload manifesting as engorged neck veins. Chest x-ray may show normal lungs or signs of blood vessel pruning. Point-of-care ultrasound (POCUS) examination of the heart shows flattening of the intraventricular septum due to right ventricular pressure overload, the D sign.

    Urine

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