◗ Why do you believe Covid is not a respiratory disease but a vascular disease?
By about January 2020, we had the first reports from China about this terrible disease. There was a high mortality rate and the mode of death, in the end, was the same: resistant to treatment, low oxygen levels. Whatever was done, be it an oxygen mask or even a ventilator, the disease was resistant to therapy and patients died.
The Chinese published small post-mortem studies on patients who had died from the original virus. Everybody was scared, so postmortems were only done on a small number of patients; one study was on nine patients; another on 13. They showed that 20–30% of the patients had macroclots – deep-vein thrombosis, clots in the lungs and so on – but 100% had microclots in the lungs.
Typically, a patient had a couple of days of fever and flu-like illness and then became short of breath, with low oxygen levels. When they presented to hospital, they would often be breathing at a rate of 30–40 breaths per minute, which is quite hard work. But they didn’t feel short of breath. So you had what we call a happy hypoxaemic patient: when the patient is comfortable but oxygen levels are low.
The functional unit of the lung is a little air sac, and if you have pneumonia, these sacs fill with pus, which causes low oxygen levels. But none of these patients was coughing up phlegm or pus; the air sac was normal. So the only thing left to cause low blood oxygen levels is a problem with the blood vessel: thrombi, blood clots. That was the original thought. And that was proven 100% by post mortem. That’s also why, initially, they found that ventilating the patient didn’t help.
◗ When you
